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In patients with cardiovascular disease, hypokalemia and even low-normal plasma potassium levels increase the risk of ventricular arrhythmias. Whether actively increasing plasma potassium to the high-normal range reduces risk is unclear.
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A population-based cohort defined risk of hyperkalemia after initiating SGLT-2 inhibitors, GLP1 receptor agonists or DPP-4 inhibitors to patients with chronic kidney disease and type 2 diabetes
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